Mawallafi: William Ramirez
Ranar Halitta: 24 Satumba 2021
Sabuntawa: 16 Nuwamba 2024
Anonim
CPK gwajin gwajin isoenzymes - Magani
CPK gwajin gwajin isoenzymes - Magani

Halittar phosphokinase (CPK) gwajin isoenzymes tana auna nau'ikan nau'ikan CPK a cikin jini. CPK enzyme ne wanda akasari aka samo shi a cikin zuciya, kwakwalwa, da kuma kasusuwa.

Ana bukatar samfurin jini. Ana iya ɗauka wannan daga jijiya. Jarabawar ana kiranta venipuncture.

Idan kana asibiti, za'a iya maimaita wannan gwajin sama da kwana 2 ko 3. Haɓakawa ko faɗuwa a cikin jimlar CPK ko CPK isoenzymes na iya taimaka wa mai ba da lafiyar ku gano wasu yanayi.

Ba a buƙatar shiri na musamman a mafi yawan lokuta.

Faɗa wa mai ba ka magani game da duk magungunan da kake sha. Wasu kwayoyi na iya tsoma baki tare da sakamakon gwaji. Magunguna waɗanda zasu iya ƙara ma'aunan CPK sun haɗa da masu zuwa:

  • Barasa
  • Amphotericin B
  • Wasu maganin sa maye
  • Hodar iblis
  • Fibrate kwayoyi
  • Statins
  • Steroid, irin su dexamethasone

Wannan jeri ba duka bane.

Kuna iya jin ɗan zafi lokacin da aka saka allurar don ɗiban jini. Wasu mutane suna jin ƙyallen kawai ko azaba. Bayan haka, ana iya samun wasu buguwa.


Ana yin wannan gwajin idan gwajin CPK ya nuna cewa duka matakin CPK ɗinku yana ɗaukaka. Binciken CPK isoenzyme na iya taimakawa gano ainihin asalin abin da aka lalata.

CPK an yi shi ne da abubuwa uku kaɗan daban-daban:

  • CPK-1 (ana kuma kiransa CPK-BB) galibi a cikin kwakwalwa da huhu
  • CPK-2 (wanda ake kira CPK-MB) ana samunsa galibi a cikin zuciya
  • CPK-3 (wanda ake kira CPK-MM) ana samunsa galibi a cikin tsokar ƙashi

Matakan CPK-1 mafi girma fiye da-al'ada:

Saboda ana samun CPK-1 galibi a cikin kwakwalwa da huhu, rauni ga ɗayan waɗannan yankuna na iya ƙara matakan CPK-1. Levelsara matakan CPK-1 na iya zama saboda:

  • Ciwon kwakwalwa
  • Raunin kwakwalwa (saboda kowane irin rauni ciki har da, bugun jini, ko zubar jini a cikin kwakwalwa)
  • Magungunan lantarki
  • Ciwon huhu na huhu
  • Kamawa

Matakan CPK-2 mafi girma fiye da-al'ada:

Matakan CPK-2 sun tashi awanni 3 zuwa 6 bayan bugun zuciya. Idan babu sauran ƙwayar tsoka ta zuciya, matakin ya hau kan awanni 12 zuwa 24 kuma ya dawo zuwa al'ada 12 zuwa 48 bayan mutuwar nama.


Levelsara matakan CPK-2 na iya zama saboda:

  • Raunin lantarki
  • Ibarfafa zuciya (ma'anar girgiza zuciya ta ma'aikatan likita)
  • Raunin zuciya (alal misali, daga hatsarin mota)
  • Kumburin ƙwayar tsoka yawanci saboda kwayar cuta (myocarditis)
  • Budewar tiyata

Matakan CPK-3 mafi girma fiye da-al'ada galibi alama ce ta rauni na tsoka ko damuwar tsoka. Suna iya zama saboda:

  • Murkushe rauni
  • Lalacewar tsoka saboda ƙwayoyi ko rashin motsi na dogon lokaci (rhabdomyolysis)
  • Ystwayar tsoka
  • Myositis (ƙashin ƙwayar tsoka)
  • Karɓar allurar intramuscular da yawa
  • Gwajin kwanan nan da gwajin aiki na tsoka (electromyography)
  • Kamawa ta kwanan nan
  • Tiyata kwanan nan
  • Motsa jiki mai nauyi

Abubuwan da zasu iya shafar sakamakon gwajin sun haɗa da maganin ƙwaƙwalwar zuciya, allurar intramuscular, aikin tiyata na baya-bayan nan, da motsa jiki mai ƙarfi da tsawan lokaci ko motsi.


Gwajin Isoenzyme don takamaiman yanayi kusan 90% daidai ne.

Creatine phosphokinase - isoenzymes; Creatine kinase - isoenzymes; CK - isoenzymes; Ciwon zuciya - CPK; Murkushe - CPK

  • Gwajin jini

Anderson JL. Searshen haɓaka ƙananan ƙananan ƙwayar cuta da rikitarwa na infarction na zuciya. A cikin: Goldman L, Schafer AI, eds. Magungunan Goldman-Cecil. 25th ed. Philadelphia, PA: Elsevier Saunders; 2016: babi na 73.

Marshall WJ, Day A, Lapsley M. Plasma sunadarai da enzymes. A cikin: Marshall WJ, Day A, Lapsley M, eds. Chemistry na Clinical. 8th ed. Philadelphia, PA: Elsevier; 2017: babi na 16.

Nagaraju K, Gladue HS, Lundberg IE. Cututtukan kumburi na tsoka da sauran ƙwayoyin cuta. A cikin: Firestein GS, Budd RC, Gabriel SE, McInnes IB, O'Dell JR, eds. Littafin Kelley da Firestein na Rheumatology. 10 ed. Philadelphia, PA: Elsevier Saunders; 2017: babi na 85.

Selcen D. Cututtukan tsoka. A cikin: Goldman L, Schafer AI, eds. Magungunan Goldman-Cecil. 25th ed. Philadelphia, PA: Elsevier Saunders; 2016: babi na 421.

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